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Category:Australian animated television series
Category:Australian children’s animated television series
Category:Australian television showsQ:

What is QT::RenderWindow?

In the Qt 4 documentation, it says this:

A QT::RenderWindow represents a visual context. Different visual contexts can be used to render the same UI on different devices.

What does this mean?

A:

You can read more about the RenderWindow class here:

RenderWindow
This is one of the base classes that allows your application to maintain
different states (state machine). It also implements sharing of device
resources, allowing the application to abstract and maintain different
states of the visual user interface. This class is designed to manage
state changes including rendering and updating the screen.

The major goal of this application is to define the mechanisms involved in the regulation of stem cell proliferation and expansion. It was first shown that adult stem cells can be expanded indefinitely using interleukin-2 (IL-2) or phorbol ester as growth stimuli. More recently, IL-2-activated T cells were used in a nonmyeloablative strategy for the treatment of cancer. While several clinical trials have been initiated with this system, the mechanisms involved in this pathway of cancer treatment are still poorly understood. It was recently found that the combination of IL-2 and phorbol ester can protect hematopoietic stem cells (HSC) from the myelotoxic effects of conventional chemotherapy. This is due to a combination of two growth effects: stem cells can be protected from drug-induced cell cycle arrest; and activation of STATs via IL-2 induces the expression of a subset of genes, which can stimulate HSC proliferation. This proposal will test the model that the molecular mechanisms of stem cell-cycle protection and stem cell growth can be studied in the setting of a genetically modified mouse in which the IL-2-induced growth effect can be isolated and studied without interference from other growth effects. Aim 1 is designed to test the hypothesis that the IL-2-induced growth signals are sufficient to protect HSC from the myelotoxic effects of chemotherape
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